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arXiv:2611.12690 · v1

Average versus expected causal effects under interference: Modeling assumptions and estimation bottlenecks

首次提交 2026-11-10 00:50
版本更新 2026-11-10 00:50
雷达观测 2026-11-10 15:22
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摘要

Causal inference under treatment interference requires assumptions to reduce the exponentially large number of potential outcomes. We develop hierarchical finite-population models for potential outcomes by separating these assumptions into three layers: interference neighborhoods, exposure mappings, and outcome models. Given an interference neighborhood and exposure mapping, we show that potential outcomes admit a nonparametric decomposition into direct, spillover, and treatment--spillover interaction components. This decomposition provides a common coordinate system for nonparametric, semiparametric, and parametric models of finite-population potential outcomes. For a given potential-outcome model, we define two classes of estimands. Average causal effects (ACEs), such as direct and total treatment effects, contrast potential outcomes under fixed treatment--exposure conditions. Expected causal effects (ECEs), such as expected direct and indirect effects, contrast expectations of potential outcomes under a specified allocation policy. For both classes, we bound the variance of the canonical Horvitz--Thompson estimator by three terms: a diagonal weight term, a term from overlapping interference neighborhoods, and a residual design-dependence term. Our upper and lower bounds show that under graph-unaware designs, nonparametric design-unbiased estimation of ACEs can require sample sizes growing exponentially in the interference degree, whereas polynomial growth can suffice for design-aligned ECEs. Under neighborhood interference, we construct graph-aware independent-set designs with variance bounds polynomial in the maximum degree for direct and total treatment effects on every graph, whereas graph cluster randomization can incur worst-case variance exponential in the degree.

作者备注:42 pages, 4 figures, 30-page appendix

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v1 · 2026-11-10 00:50 · 最新观测版本

Average versus expected causal effects under interference: Modeling assumptions and estimation bottlenecks

Causal inference under treatment interference requires assumptions to reduce the exponentially large number of potential outcomes. We develop hierarchical finite-population models for potential outcomes by separating these assumptions into three layers: interference neighborhoods, exposure mappings, and outcome models. Given an interference neighborhood and exposure mapping, we show that potential outcomes admit a nonparametric decomposition into direct, spillover, and treatment--spillover interaction components. This decomposition provides a common coordinate system for nonparametric, semiparametric, and parametric models of finite-population potential outcomes. For a given potential-outcome model, we define two classes of estimands. Average causal effects (ACEs), such as direct and total treatment effects, contrast potential outcomes under fixed treatment--exposure conditions. Expected causal effects (ECEs), such as expected direct and indirect effects, contrast expectations of potential outcomes under a specified allocation policy. For both classes, we bound the variance of the canonical Horvitz--Thompson estimator by three terms: a diagonal weight term, a term from overlapping interference neighborhoods, and a residual design-dependence term. Our upper and lower bounds show that under graph-unaware designs, nonparametric design-unbiased estimation of ACEs can require sample sizes growing exponentially in the interference degree, whereas polynomial growth can suffice for design-aligned ECEs. Under neighborhood interference, we construct graph-aware independent-set designs with variance bounds polynomial in the maximum degree for direct and total treatment effects on every graph, whereas graph cluster randomization can incur worst-case variance exponential in the degree.

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